Mr. Armali is a 53-year-old male who comes to the clinic with concerns over his blood pressure. He has been checking it at home and it ranges from 160-188/86-100. In the clinic today, his current blood pressure is 172/98. He has no complaints of headache or chest pain. You plan to start him on an angiotensin-converting enzyme inhibitor (ACEI) and diagnose him with primary hypertension.
Discuss the pathophysiology of primary hypertension and the pathologic effects associated with it.
- Minimum of 250 words (one double-spaced page is sufficient). There is no need for a lengthy introduction or conclusion.
- 2 scholarly sources within the last 5 years (one may be your textbook).
- APA 7th edition Student Format
- Insert hyperlinks in your reference page
- All discussion posts must be submitted through Turnitin. Any post with more than a 20% similarity score will have one opportunity to revise and resubmit their work.
- If this week’s discussion post consists of a case study, you may mention the case studies in your answer to the question(s), but do not write them verbatim. It will not count toward your word count.
Primary hypertension is a chronic and progressive condition that is characterized by sustained elevated blood pressure levels. It is caused by a combination of several factors, including genetic predisposition, lifestyle factors, and environmental factors. The pathophysiology of primary hypertension is not fully understood. However, it is believed to involve the constriction of blood vessels, increased salt and water retention in the kidneys, and increased secretion of certain hormones. The primary pathologic effect of primary hypertension is the increased workload on the heart, leading to increased risk of coronary artery disease, stroke, and congestive heart failure. The increased pressure in the blood vessels can also lead to damage of the walls of the blood vessels, leading to aneurysm formation and other vascular problems. In addition, the increased pressure in the vessels can lead to leakage of fluid from the blood vessels into surrounding tissue, causing edema and organ damage. Finally, the increased pressure in the vessels can lead to a decrease in the supply of oxygen and nutrients to the organs, leading to organ failure.
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